ECT2L
-
Official Full Name
epithelial cell transforming sequence 2 oncogene-like -
Synonyms
ECT2L;epithelial cell transforming sequence 2 oncogene-like;21118;ENSG00000203734;LFDH;FBXO49;C6orf91;ARHGEF32;dJ509I19.2;dJ509I19.3;dJ509I19.5;6q24.1;epithelial cell-transforming sequence 2 oncogene-like;epithelial cell-transforming sequence 2 oncogene-like;ECT2-like;F-box protein 49;putative guanine nucleotide exchange factor LFDH;lung specific F-box and DH domain containing protein;lung-specific F-box and DH domain-containing protein
Cat.# | Product name | Source (Host) | Species | Tag | Protein Length | Price |
---|
Involved Pathway
ECT2L involved in several pathways and played different roles in them. We selected most pathways ECT2L participated on our site, such as , which may be useful for your reference. Also, other proteins which involved in the same pathway with ECT2L were listed below. Creative BioMart supplied nearly all the proteins listed, you can search them on our site.
Pathway Name | Pathway Related Protein |
---|
Protein Function
ECT2L has several biochemical functions, for example, . Some of the functions are cooperated with other proteins, some of the functions could acted by ECT2L itself. We selected most functions ECT2L had, and list some proteins which have the same functions with ECT2L. You can find most of the proteins on our site.
Function | Related Protein |
---|
Interacting Protein
ECT2L has direct interactions with proteins and molecules. Those interactions were detected by several methods such as yeast two hybrid, co-IP, pull-down and so on. We selected proteins and molecules interacted with ECT2L here. Most of them are supplied by our site. Hope this information will be useful for your research of ECT2L.
Resources
Related Services
Related Products
References
- Wang, HX; Sharma, C; et al. EWI-2 negatively regulates TGF-beta signaling leading to altered melanoma growth and metastasis. CELL RESEARCH 25:370-385(2015).
- Dommisch, H; Winter, J; et al. Effect of growth factors on antimicrobial peptides and pro-inflammatory mediators during wound healing. CLINICAL ORAL INVESTIGATIONS 19:209-220(2015).