CD2A
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Official Full Name
CD1A CD2a molecule -
Overview
CD1a (Cluster of Differentiation 1a) is a human protein encoded by the CD1A gene. This gene encodes a member of the CD1 family of transmembrane glycoproteins, which are structurally related to the major histocompatibility complex (MHC) proteins and form h -
Synonyms
CD1A;R4;T6;CD1;FCB6;HTA1;T-cell surface glycoprotein CD1a;OTTHUMP00000018907;hTa1 thymocyte antigen;CD1A antigen, a polypeptide;cluster of differentiation 1 A;T-cell surface antigen T6/Leu-6;cortical thymocyte antigen CD1A;differentiation antigen CD1-alpha-3;epidermal dendritic cell marker CD1a
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Involved Pathway
CD2A involved in several pathways and played different roles in them. We selected most pathways CD2A participated on our site, such as , which may be useful for your reference. Also, other proteins which involved in the same pathway with CD2A were listed below. Creative BioMart supplied nearly all the proteins listed, you can search them on our site.
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Protein Function
CD2A has several biochemical functions, for example, . Some of the functions are cooperated with other proteins, some of the functions could acted by CD2A itself. We selected most functions CD2A had, and list some proteins which have the same functions with CD2A. You can find most of the proteins on our site.
| Function | Related Protein |
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Interacting Protein
CD2A has direct interactions with proteins and molecules. Those interactions were detected by several methods such as yeast two hybrid, co-IP, pull-down and so on. We selected proteins and molecules interacted with CD2A here. Most of them are supplied by our site. Hope this information will be useful for your research of CD2A.
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References
- Bugelski, PJ; Achuthanandam, R; et al. Monoclonal antibody-induced cytokine-release syndrome. EXPERT REVIEW OF CLINICAL IMMUNOLOGY 5:499-521(2009).
- Xu, Y; Simonds, DK; et al. The anti-CD2 monoclonal antibody BTI-322 generates unresponsiveness by activation-associated T cell depletion. CLINICAL AND EXPERIMENTAL IMMUNOLOGY 138:476-483(2004).
